Establishing marmoset models of Alzheimer’s Disease to identify emerging phenotypes and illuminate mechanisms underlying pathogenesis.
Overview:
Marmosets bridge the rodent to human translational gap
Overcome limitations of other model organisms by exhibiting primate-specific aging and dementia phenotypes
Gain translatable knowledge through simultaneous assessment of genetic, molecular, functional, behavioral, and pathological phenotypes
Identify emerging phenotypes that precede frank neuropathy through gene-edited models with genetic risk for EOAD and LOAD
Goals:
Establish the marmoset as the first primate-specific model of AD
Reveal the earliest cellular and molecular events of AD processes and allow charting AD progression from its inception.
Develop marmoset models of early-onset AD and late-onset AD
Investigate the underlying cellular and molecular root causes of the pathogenesis and progression of AD.
Assess genetic, molecular, functional, behavioral, and pathological phenotypes simultaneously in marmosets.
Provide translatable knowledge of the origins and progression of AD in human populations
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